Abel – Case Study 1 of 6 Recognize Cues For each finding, select whether it is a clinical cue for PUD with upper GI bleeding or not.
Rationale
Right facial droop + arm drift → ✅ Clinical cue — hallmark unilateral FAST signs confirming hemispheric involvement
Dysarthria + confusion → ✅ Clinical cue — motor speech dysfunction + reduced cerebral perfusion = cortical involvement
Sudden headache × 2 hrs → ✅ Clinical cue — cardinal stroke warning; establishes thrombolytic eligibility window
Right leg weakness → ✅ Clinical cue — extensive unilateral motor deficit = significant hemispheric stroke
DVT history → ✅ Clinical cue — increases thromboembolic risk via paradoxical embolism and hypercoagulability
BP 150/90 + HR 110 → ✅ Clinical cue — HTN = #1 modifiable stroke risk; tachycardia = stress response to neuro injury
SpO₂ 95% → ✅ Clinical cue — hypoxia worsens ischemic injury to penumbral tissue
Glucose 112 mg/dL → ❌ Not a cue — rules out hypoglycemia as stroke mimic only; not diagnostic for stroke
Right facial droop + arm drift → ✅ Clinical cue — hallmark unilateral FAST signs confirming hemispheric involvement
Dysarthria + confusion → ✅ Clinical cue — motor speech dysfunction + reduced cerebral perfusion = cortical involvement
Sudden headache × 2 hrs → ✅ Clinical cue — cardinal stroke warning; establishes thrombolytic eligibility window
Right leg weakness → ✅ Clinical cue — extensive unilateral motor deficit = significant hemispheric stroke
DVT history → ✅ Clinical cue — increases thromboembolic risk via paradoxical embolism and hypercoagulability
BP 150/90 + HR 110 → ✅ Clinical cue — HTN = #1 modifiable stroke risk; tachycardia = stress response to neuro injury
SpO₂ 95% → ✅ Clinical cue — hypoxia worsens ischemic injury to penumbral tissue
Glucose 112 mg/dL → ❌ Not a cue — rules out hypoglycemia as stroke mimic only; not diagnostic for stroke
